Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.
Answer:
Explanation:
We are asked to find the energy given mass, specific heat, and change in temperature. Therefore, we must use this formula;
The mass is 15 grams and the specific heat is 0.129 J/(g×°C). Let's calculate the change in temperature.
- ΔT= final temperature - initial temperature
- ΔT= 85 °C- 22°C = 63°C
Now we know all the values:
Substitue the values into the formula.
Multiply the first numbers together. The grams will cancel.
Multiply again, this time the degrees Celsius cancels.
<u>121.905 Joules</u> are required.
The initial stage of atherosclerosis is characterized by accumulation beneath the endothelium of excessive amounts of LDL.
- The accumulation of lipids, cholesterol, and other chemicals in and on the artery walls is known as atherosclerosis. The accumulation is known as plaque.
- The build-up of lipids and fibrous components in the major arteries is a hallmark of the degenerative illness known as atherosclerosis. A picture of a typical artery's anatomy can be found in Foam cells, or subendothelial accumulations of cholesterol-engorged macrophages, are the earliest lesions of atherosclerosis.
- Nitric oxide and prostacyclin are two chemicals that are produced by your endothelium. These maintain the fluid in your blood and stop it from clotting when it shouldn't. These compounds' insufficient production is a symptom of various diseases, which raises your risk of clotting.
To learn more about atherosclerosis.
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