Answer:
Helicase
Explanation:
The DNA double helix is unzipped by the enzyme *HELICASE*. The enzyme breaks the hydrogen bond between the bases thereby causing the strands of the double helicase to unzip. The action of the helicase enzyme uses energy from ATP. The unzipping of the double helix does not occur in a swift move. One end of the DNA molecule with 5'-3' is opened up showing the antiparallel single strands. The end that is opened is termed INITIATION SITE of replication. The opening look like someone standing with legs wide open. The wide opening is called REPLICATION FORK. The single strands of the fork are usually unstable but their stability is maintained by the DNA-binding-proteins. Having opened the double helix, the replication would then continue in the 5'-3' direction of one of the two antiparallel single strands.
I think its A because the small size , shape and the methods of movement have nothing to do with the environments it can survive in <span />
Answer:
The tendency of water molecules to stick to other molecules also allows water to dissolve substances that are important to life, such as ions, DNA, and many proteins. This is necessary for life because molecules that have been dissolved in water or another polar solvent interact with each other more readily.
Answer:
<u> The following four traits are -: </u>
- <u>Pedigree 1 -</u> A recessive trait (autosomal recessive) is expressed by pedigree 1.
- <u>Pedigree 2- Recessive inheritance is defined by Pedigree 2. </u>
- <u>Pedigree 3</u> - The inheritance of the dominant trait (autosomal dominant) is illustrated by Pedigree 3.
- <u>Pedigree 4-</u> An X-like dominant trait is expressed by Pedigree 4.
Explanation:
<u>Explaination of each pedigree chart</u>-
- Pedigree 1 demonstrates the <u>recessive trait </u>since their children have been affected by two unaffected individuals. If the characteristics were X-linked, in order to have an affected daughter, I-1 would have to be affected.
In this, both parents are autosomal recessive trait carriers, so the child will be affected by a 1/4 (aa) - <u> Recessive inheritance</u> is defined by <u>Pedigree 2</u>. This is<u> X-related inheritance as autosomal recessive</u> inheritance has already been accounted for in part 1. This inference is confirmed by evidence showing that the father (I-1) is unaffected and that only the sons exhibit the characteristic in generation II, suggesting that the mother must be the carrier. The individual I-2 is a carrier for this X-linked trait. A typical Xa chromosome is attached to the unaffected father (I-1), so the chance of carrier II-5 is 1/2. Probability of an affected son = 1/2 (probability II-5 is a carrier) x 1/2 (probability II -5 contributes (
) x 1/2 (probability of Y from father II-6) = 1/8. An affected daughter's likelihood is 0 because a typical
must be contributed by II-6. - The inheritance of the<u> dominant trait</u> is demonstrated by <u>Pedigree 3 </u>because affected children still have affected parents (remember that all four diseases are rare). The trait must be <u>autosomal dominant</u> because it is passed down to the son by the affected father. There is a 1/2 risk that the heterozygous mother (II-5) would pass on mutant alleles to a child of either sex for an autosomal dominant feature.
- <u>Pedigree 4</u> is an <u>X-linked dominant function</u> characterized by the transmission to all of his daughters from the affected father but none of his son. On the mutant X chromosome, the father (I-1) passes on to all his daughters and none of his sons. As seen by his normal phenotype, II-6 therefore does not bear the mutation. An affected child's likelihood is 0.
In the question the pedigree chart was missing ,hence it is given below.