To find an angle you use a variable like x or y. Sincw its a straight angle, it would be x +103 = 180. then you would subtract 103 on both sides. then you'll get x= 77 degrees
Reduce osteoblast activity while boosting osteoclast activity
osteoblast:
- Untreated celiac disease has a number of potential side effects, including the failure for children to reach their ideal bone density and adult bone loss, both of which raise the risk of osteoporosis. Following a gluten-free diet is the only way to treat celiac disease.
- As a result, celiac disease is common in both children and adults with low bone density. When celiac disease is discovered in an adult, the risk of osteoporosis is very significant (because they have gone longer without absorbing enough calcium and other nutrients).
- Osteoporosis is a bone disease that appears when bone mass, bone mineral density, or both drop or when the quality or structure of the bone deteriorates. This may result in a loss of bone density and an elevated risk of fractures (broken bones).
Learn more about osteoporosis here brainly.com/question/14601010
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<span>There are numerous proteins in muscle. The main two are thin actin filaments and thick myosin filaments. Thin filaments form a scaffold that thick filaments crawl up. There are many regulatory proteins such as troponin I, troponin C, and tropomyosin. There are also proteins that stabilize the cells and anchor the filaments to other cellular structures. A prime example of this is dystrophin. This protein is thought to stabilize the cell membrane during contraction and prevent it from breaking. Those who lack completely lack dystrophin have a disorder known as Duchene muscular dystrophy. This disease is characterized by muscle wasting begininng in at a young age and usually results in death by the mid 20s. The sarcomere is the repeating unit of skeletal muscle.
Muscle cells contract by interactions of myosin heads on thick filament with actin monomers on thin filament. The myosin heads bind tightly to actin monomers until ATP binds to the myosin. This causes the release of the myosin head, which subsequently swings foward and associates with an actin monomer further up the thin filament. Hydrolysis and of ATP and the release of ADP and a phosphate allows the mysosin head to pull the thick filament up the thin filament. There are roughly 500 myosin heads on each thick filament and when they repeatedly move up the thin filament, the muscle contracts. There are many regulatory proteins of this contraction. For example, troponin I, troponin C, and tropomyosin form a regulatory switch that blocks myosin heads from binding to actin monomers until a nerve impulse stimulates an influx of calcium. This causes the switch to allow the myosin to bind to the actin and allows the muscle to contract. </span><span>
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Have a lot of mitochondria so that more respiration providing muscles with more energy for muscle contraction
Hope it’s useful
