The correct answer is the last statement.
If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.
It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.
In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.
Autoinducer molecules are signalling molecules that need bacteria. In order for the autoinducer molecules to signal, the bacteria must find a way to initiate the signal and a way to respond to the signal it had created. Autoinducer molecules increase in production as the density of the bacteria cell increases.
Answer:
1. they come mainly in 2 parts (Felis catus is for a domestic cat) = all in the binomal naming system format
2. they are all in the same language so anyone in the world can interpret what animal it is= latin
3. genus and species= this means that the first scientific name (ie Felis) is the genus and Catus is the species of the feline organisms
Explanation:
Answer:
D
Explanation:
Nutrition wise euglena is a producer, but locomotion wise it is classified as animal. Other example is chlamydomonas
