Answer:
The main function of the RB protein is to inhibit the transition from the G1 to S phase. Its activity depends on its phosphorylation state: if RB is not phosphorylated (active state), it is bound to the transcription factor E2F, preventing its translocation to the nucleus and the activation of genes necessary for DNA synthesis; and if it is phosphorylated, by cyclin-CDK complexes, E2F is released and cell proliferation occurs.
Explanation:
Retinoblastoma (Rb) was the first tumor suppressor described and, as we know today, mutations in its structure determine a large number of cancers. Structurally, we speak of a large and multifunctional protein; which is organized in different domains. The activity of this protein is regulated, mainly, by phosphorylation at multiple sites described over the years and is involved in control mechanisms of the cell cycle, apoptosis and senescence.
Answer:
The word that best complete the sentence is VIRUSES.
Explanation:
There are different types of microorganisms, one of them is virus. Viruses are different from other forms of microbes because they need living cells to become active; outside living cells they are inactive and did not exhibit any of the features of living things.
Viruses only grow and multiply when they are withing the cells of another living thing; they do not have the capacity to survive and reproduce outside a living host.
Answer:
a plant hormone which promotes leaf detachment, induces seed and bud dormancy, and inhibits germination.
Answer:
true
Explanation:
Daylight hours are shortest in each hemispheres winter. between summer and Winter Solstice, the number of daylight hours decreases, and the rate of decrease is larger the higher latitude. the fewer sunlight hours the colder the nights.
The question is incomplete as it does not have the options which are:
photoreactivation
reactivation repair pathway
nucleotide excision repair pathway
UV repair pathway
p53 repair pathway
Answer:
p53 repair pathway
Explanation:
ATM gene or ataxia telangiectasia-mutated is a gene which codes for the protein kinase involved in the DNA double-stranded break.
The DNA double-stranded break is repaired easily as the protein kinase is recruited to the site of the break where it allows the DNA repair machinery to repair the DNA.
The gene also controls the cell growth therefore this gene is important to study cancer. Since the p53 protein is a suppressor protein which if mutated is repaired by the ATM gene and if the gene becomes mutated then the p53 repair pathway is affected.
Thus, p53 repair pathway is the correct answer.
