Answer:
Explanation:
Amphibians are group of animals that live a double life; one in water with gills and the other on land with lungs (at maturity). They include frogs, newts, salamanders etc.
Reptiles ( meaning to creep under shadow of darkness) are group of animals that possess scales. They include snakes, crocodiles, lizards etc.
Reptiles and amphibians are distantly related but share some traits including:
1. They are both poikilothermic (cold-blooded) animals i.e. their body temperature is dependent on that of the environment's.
2. They both lay eggs externally, although that of amphibians is fertilized after being layed
3. They both possess lungs for gaseous exchange, although, amphibians use gills in water.
4. Both reptiles and amphibians belongs to Chordata i.e. they both possess a vertebrae column (backbone)
5. Most reptiles and amphibians make use of the same defensive and attacking traits as a pretty and predator respectively. They employ camouflage, biting and inflating of their body size.
I think it’s C I hope it’s right I’m sorry if I’m wrong
Death receptors (SDRs) result in apoptosis however additionally stimulate proinflammatory "non-apoptotic" signaling (e.g. NF-κB and mitogen-activated protein kinase (MAPK) activation) and inhibit awesome steps of DR-activated maturation of procaspase-8. To take a look at whether or not isoforms of cellular FLIP (cFLIP) or its cleavage products differentially regulate DR signaling, we mounted HaCaT cells expressing cFLIP(S), cFLIP(L), or mutants of cFLIP(L) (cFLIP(D376N) and cFLIP(p43)). cFLIP editions blocked TRAIL- and CD95L-induced apoptosis, but the cleavage pattern of caspase-8 in the dying inducing signaling complicated was different: cFLIP(L) brought on the processing of caspase-8 to the p43/41 fragments irrespective of cFLIP cleavage. cFLIP(S) or cFLIP(p43) blocked procaspase-8 cleavage. Analyzing non-apoptotic signaling pathways, we found that TRAIL and CD95L activate JNK and p38 within 15 min. cFLIP variations and exclusive caspase inhibitors blocked late demise ligand-induced JNK or p38 MAPK activation suggesting that these responses are secondary to mobile death. cFLIP isoforms/mutants also blocked dying ligand-mediated gene induction of CXCL-8 (IL-8). Knockdown of caspase-8 completely suppressed apoptotic and non-apoptotic signaling. Knockdown of cFLIP isoforms in most important human keratinocytes improved CD95L- and TRAIL-induced NF-κB activation, and JNK and p38 activation, underscoring the regulatory position of cFLIP for these DR-mediated signals. Whereas the presence of caspase-8 is fundamental for apoptotic and non-apoptotic signaling, cFLIP isoforms are strong inhibitors of TRAIL- and CD95L-induced apoptosis, NF-κB activation, and the late JNK and p38 MAPK activation. cFLIP-mediated inhibition of CD95 and TRAIL DR could be of necessary importance for the duration of keratinocyte skin carcinogenesis and the activation of innate and/or adaptive immune responses induced using DR activation in the skin.
Any of two or greater functionally comparable proteins that have a similar but not same amino acid sequence and are either encoded by means of extraordinary genes or with the aid of RNA transcripts from the identical gene which have had special exons removed.
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Answer:
In creating this list, one will have to start from the atom (which is the smallest in the list) to the giant polymer (which is the largest in the group). The arrangement below will also be based on there molecular weight.
oxygen molecule (this is the first because it is an atom)
water molecule (this is lighter in weight than carbon dioxide)
carbon dioxide
amino acid molecule (most amino acids have a lesser molecular weight than glucose)
glucose (has a molecular weight of 180.156 g/mol)
protein molecule (several proteins have a lesser molecular weight than starch)
starch molecule (has a molecular weight of 359.33 g/mol)
Explanation:
Answer:
Global climate models usually work by managing the planet as a set of connected boxes because this facilitates the management of the calculations applied in a climate system that is dynamic, variable and complex, which is due to the fact that there is not an equal pattern of climate in each area of the planet.
Explanation:
Climate models have been designed to show the dynamics of the planet's climate and what can be expected from the changes that occur in the future.
The scientific basis of a climate model is physical phenomena and chemical reactions to which a series of differential calculations are applied to determine the behaviour of:
- <em>Winds.</em>
- <em>Temperature.</em>
- <em>Relative humidity.</em>
- <em>Rainfall.</em>
- <em>Pressure changes.</em>
- <em>Heat transfer.</em>
- <em>Hydrological dynamics.
</em>
Global climate models usually divide the planet into sections -such as boxes- making the calculations in each sector, as well as the interaction of each section with the adjacent sections, which greatly facilitates making the differential calculations, showing the climate at a given time and making predictions of future climate behavior.
It is important to mention that a climate model also explains the changes that occur in the climate due to human activity, which is different in each region of the world.
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Global climate models brainly.com/question/9423898
