Answer:
Dominant
Explanation:
I'm pretty sure of this. C is dominant and that out weighs recessive.
MRNA, ribsomes,tRNAs, and many enzymatic factors
Answer:a. Draw Punnett squares for each couple (you may need to do more than 1 square/ couple)
Baby 2 MUST belong to the Browns because Mr. Brown is the only parent with an A allele to
contribute… then the rest works out as follows:
b. To which parents does baby #1 belong? Why? Hint you may want to refer to your Punnett
squares.
Baby 1 must belong to the Smiths, because they are the only ones with the possibility of EACH
having a recessive allele to pass down to the baby, Mr. Brown has type AB blood and therefore
only has the dominant A and dominant B alleles – no recessive allele possible.
Explanation:
Answer:
The correct insulin pathway is described as under:
2. Binding of insulin to the alpha subunit of the insulin receptor
8. Activation of insulin receptor tyrosine kinase
3. Phosphorylation of IRS proteins
6. Phosphorylation of phosphinositide 3-kinase (PI-3K)
4. Conversion of PIP2 to PIP3
7. Activation of PIP3-dependent protein kinase B (PDK1)
5. Glut4 receptors transported to the cell membrane
Explanation:
The insulin signaling pathway is described as under:
RTK (receptor tyrosine kinases) which is a receptor for insulin is an extracellular receptor but in contrast to other cell surface receptors it is catalytic in nature. In the absence of insulin (ligand), it is monomeric but as soon as it gets activated (activation occurs upon ligand binding), it undergo dimerization. It leads to auto-phosphorylation in it's tyrosine residue which subsequently leads to phosphorylation of tyrosine residue of other receptors. Such hyper-phosphorylated receptor have high affinity with enzyme/molecule like IRS protein which have SH2 domain . IRS down stream activates phosphinositide 3-kinase (PI-3K). This enzyme converts component of animal cell membrane PIP2 into PIP3. PIP3 also remains membrane bound but it has the potential to phosphorylate another enzyme named as PIP3-dependent protein kinase B (PDK1). Further, PDK1 leads to the activation of Akt or PK-B. Akt is a serine-threonine kinase which ultimately leads to the recruitment of Glut4 receptors on cell membrane for uptake of more and more glucose into the cell.
Note: Apart from this Akt also phosphorylates another protein named as FOXO which ultimately causes cell growth, Akt can also phosphorylate BAD protein so as to restrict cell apoptosis or we can say it leads to cell survival, Akt also leads to translation in a cell with the help of mTOR raptor etc.
Answer:
Base substitution mutation
Explanation:
Mutation is the alteration or change that occurs to the sequence of a gene caused by a mutagen (chemicals) or spontaneously-induced. Depending on how the mutation occurs, it is of different types. The mutation shown in this question is as follows:
ATCGGCTTAACG to ATCGGCTAAACG
This type of mutation is called SUBSTITUTION MUTATION. Substitution mutation is a type of mutation that involves the replacement of one or more nucleotide base by another. In this original sequence; ATCGGCTTAACG, the eight base T was replaced by base A in the sequence, hence, it is a substitution mutation.
