Answer:
No, telomerase is not an oncogene. It prevents the senescence that would occur due to shortened telomeres, but the cell proliferation might still be mitogen-dependent.
Explanation
telomerase is not responsible for causing deregulation while oncogenes cause deregulation .
Telomeres length shorten after the cell division which stops them to divide again and cell die.
Telomerase prevents this decline in some kinds of cells, including stem cells, by lengthening telomeres, and the hope was that activating the enzyme could prevent senescence.
Answer:
pointing up labeled 3 N, an arrow pointing right labeled 2 N, an arrow pointing
Explanation:
Answer: Protein folding and oligomerization
Explanation:
Binding immunoglobulin protein (BiP) is a vital protein present in humans essential for the translocation of secreted peptides.
BiP is a molecular chaperone which is present in lumen of ER (endoplasmic reticulum) which binds to the new protein and then translocat into the ER. The protein in ER is maintained under subsequent condition and important for protein folding and oligomerization (conversion of a monomer or group of monomer into an oligomer).
Several other functions of BiP are:
- ER translocation
- ER-associated degradation (ERAD)
- UPR pathway
Hence, BiP is a chaperone, it is important for protein folding and oligomerization.
