When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
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Answer:
C. K- Strategy
Explanation:
this type of strategy involves greater investment from parents into their offspring.
Correct answer: Option D- DNA ligase
Explanation: In option A, thymine is a nucleotide, so it is present throughout the replication process, wherever it is required. It is added to the newly formed DNA. In option B, Helicase enzyme is active during initiation and elongation stage, as it facilitates the opening of the winded DNA strands. Option C is nucleotidase and it has no role in DNA replication. So, the correct answer is DNA ligase, which is option D.
The okazaki fragments formed during DNA replication are sealed at the end. And in this step, DNA ligase is used. It catalyzes the formation of phosphodiester bond between the nucleotides of okazaki fragments. So it is the last active molecule of the process.
Your answer for this problem is going to be :T-A-A-G-U-T-U-G
Answer: Peripheral System
Explanation:
