ATP (adenosine triphosphate)
The complementary pair would be 3' TACCCCGCG 5'
I hope I helped!
Answer:
The best answer to the question: The preferential use of phosphatidylinositol and phosphatidylethanolamine in the cytoplasmic leaflet of membranes leads to increased:___ would be, D: All of the answers are correct.
Explanation:
There is still a lot of debate among scientists as to the exact way in which phospholipids are distributed between the two leaflets of a cell´s plasma membrane, but what they do know is that there are four kinds, and that phosphatidylinositol (a sterol) and phosphatidylethanolamine appear most commonly on the cytosolic, or cytoplasmic side of the membrane. They also believe that it is this particular arrangement which allows the cytoplasmic side of the membrane to be slightly negatively charged and this role will play a major part in a membrane´s ability to generate an action potential. Also, it is known that these negatively charged ends of the bilayer will allow certain proteins, positively charged ones, to be inserted, and connected to it, to become transporters, or mediators in action potentials and normal activities of cells. Finally, since fatty acids and cholesterol are associated with phospholipids, but have a tendency to be attracted to these particular two types, they tend to give the overall structure fluidity, but also permit the inner leaflet to assume its particular circular shape around the cytoplasm. This is why the answer is D.
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
