Yes because you have a smaller population
Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.
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The answer is C. Golgi Apparatus
A sacromere is a segment between two adjacent Z discs and are essential for the striated structure of the cardiac and skeletal muscles.
<u>Explanation:</u>
The Z disc is surrounded by the I band made of thin filament called actin. The I band is followed by the A band made up of thick filament called myosin. When the muscles contract the actin and the myosin become superimposed/overlapped.
The sliding filament model explains the contraction of the sacromere in which the Z discs move closer due to the overlapping of the thin and thick filaments. Thus the I band moves close to the A band which remain the same length as shown in figure.
