Answer:
Spend more time outdoors, eat more red meat, supplement their deficiency with vitamins
Explanation:
Hello!
Vitamin D is a very important micronutrient within our bodies. This is due to its ability to regulate the amount of calcium and phosphate in the body. If a patient is Vitamin D deficient it can lead to a magnitude of problems such as bone deformities such as rickets, significant loss in bone density and depression. Thankfully, Vitamin D is one of the easiest vitamins to obtain. If a patient is not at risk for Melanoma, or other forms of skin diseases, they can obtain Vitamin D by simply spending more time outdoors in direct sunlight. Vitamin D can also be obtained through altering ones diet to include more red meat, egg yolks, liver, fish, and select breakfast cereals. If these options are not applicable to a patient a healthcare physician can supplement them in the form of Vitamin D tablets of varying concentration.
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H.M.
Answer:tools
Explanation:because we use them so it can help us
If he is trying to consume 30% of his calories from fat, he should consume 33 grams of fat.
<h3>How much fat should I consume per day?</h3>
The WHO recommendation is to consume up to 30% fat of the total calories ingested per day. That's considering a diet of 2000 kcal/day.
With this information, we can conclude that if he is trying to consume 30% of his calories from fat, he should consume 33 grams of fat.
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Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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