Answer:
The virulence factors of a pathogenic microorganism allow it to more easily invade the tissues of an organism or of a living being. In addition, it gives them greater survival power in extreme environments and unfavorable conditions for their metabolism.
Explanation:
An example of a virulence factor is the presence of flagella or capsules in bacteria, the capsules allow it to sustain life at extreme temperatures and conditions, and the flagella facilitate their mobilization. On the other hand, there are also some microorganisms that have toxins (endo or exotoxins) that work as extractors of human immunity or as destroyers of tissues. In order to achieve its greatest proliferation and survival within a living organism.
Virulence factors are also considered today as those resistance that microorganisms acquire to certain drugs or antiseptics, etc.
D, three to five times a week.
Calcium is an important element required for muscle contraction and bone integrity.
Systemic lupus erythematosus (SLE) is a rheumatic disease attributed to autoimmune mechanisms.
Systemic lupus erythematosus (SLE, lupus) is characterized by a global loss of self-tolerance with activation of autoreactive T and B cells, leading to the production of pathogen autoantibodies and tissue damage.
Innate immune mechanisms are required for aberrant adaptive immune responses in SLE. Recent advances in basic and clinical biology have shed new light on the disease mechanisms of lupus. This review article describes recent studies that provide valuable insights into disease-specific therapeutic targets.
Systemic lupus erythematosus (SLE or lupus) is a systemic autoimmune disease with inflammation of multiple organs. SLE is characterized by the production of pathogenic autoantibodies against nucleic acids and their binding proteins, reflecting a global loss of self-tolerance.
Loss of tolerance with subsequent immune dysregulation is the result of genetic factors in the context of environmental triggers and stochastic events, with recent studies implicating more than 30 genetic loci in disease pathogenesis.
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