Answer:
False, chemical energy is not transfered or converted into cellular energy
Explanation:
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
Answer:
Nitrogen in its gaseous form (N2) can’t be used by most living things. It has to be converted or ‘fixed’ to a more usable form through a process called fixation. Biologically: Nitrogen gas (N2) diffuses into the soil from the atmosphere, and species of bacteria convert this nitrogen to ammonium ions (NH4+), which can be used by plants.
Explanation:
Nutrients entering a cell through the cell membrane Removing a phosphate group from an ATP molecule Building sugar or starch molecules for long-term energy storage Adding an additional phosphate group to an ADP molecule.
Answer
D)A proton gradient alone without a corresponding energy input is not sufficient to drive the synthesis of ATP
Explanation:
Oxidative phosphorylation reffers to metabolic pathway whereby enzyme is used by the cell to achieve nutrient oxidation, so that chemical energy of molecular oxygen is released as a result of this, adenosine triphosphate (ATP) is manufactured. oxidative phosphorylation is seen in all organisms that are aerobic in nature and it occur in the mitochondria of all eukaryotic organisms. There is electron transfer during oxidative phosphorylation, the electrons moves from the donor to the receptor and this is known as redox reaction, as a result of this energy is released, and this energy is used for formation of ATP.
Therefore,among all the options, A proton gradient alone without a corresponding energy input is not sufficient to drive the synthesis of ATP is the false statement.
