Answer:Biological structures are able to adapt their growth to external mechanical stimuli and impacts. For example, when plants are under external loads, such as wind force and self-weight, the overloaded zones are reinforced by local growth acceleration and the unloaded zones stop growing or even shrink. Such phenomena are recorded in the annual rings of trees. Through his observation of the stems of spruce, K. Metzger, a German forester and author, realized that the final goal of the adaptive growth exhibited by biological structures over time is to achieve uniform stress distribution within them. He published his discovery in 1893.12 A team of scientists at Karlsruhe Research Centre adopted Metzger's observations and developed them to one single design rule: the axiom of uniform stress. The methods derived from this rule are simple and brutally successful like nature itself. An excellent account of the uniform-stress axiom and the optimization methods derived from it is given by Claus Mattheck in his book ‘Design in Nature’.13 The present study utilizes one of these methods, stress-induced material transformation (SMT), to optimize the cavity shape of dental restorations.
Explanation:
Digested food that is not need and is excreted out of the anus. Known as poop. Another waste is water that is not needed or it was already used and is let out of the penis or <span>Vagina</span>.
Answer:
Antibiotic resistance is when pathogens such as bacteria or fungi develop a resistance to antibiotics. This happens when some pathogens die, and others survive. The small number of pathogens that had a resistance to the antibiotic will reproduce more offspring that are also resistant to, and those offspring will reproduce even more offspring and so on. Eventually most pathogens will be resistant to the antibiotic, so antibiotics won't work.
Explanation:
While anatomy deals with the structure of the parts of an organism, physiology studies the way that the parts function together.
Numerous antiepileptic medications, such phenytoin, have been designed to block voltage-gated sodium channels (VGSC) in neuronal membrane. In addition, multiple toxins and pharmacological modulators work by attaching to various biophysical states of the VGSC to cause their effects. Depending on how modulatory agents act, some VGSC states are stabilized or destabilized, altering the channel's biophysical properties. The first anticonvulsant to successfully treat epileptic disorders without causing undesirable side effects such as brain drowsiness was phenytoin.
Phenytoin has been indicated to block high-frequency neuronal activity potentials from the inner vestibule of the pore, as demonstrated by electrophysiological research and site-directed mutation.
Frequency and voltage both affect phenytoin binding.
There are theories that phenytoin interferes with the late sodium current that sustains depolarizations in epilepsy by blocking non-inactivated channels.
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