Answer:
358,800N
Explanation:
F(orce) = mass × a(cceleration)
Found it online while looking for the same question lol
<span>A prokaryote is a unicellular organism that belongs to either the domain Bacteria or the domain Archaea. Therefore, unlike a eukaryote, the prokaryote lacks in a nucleus and other organelles. The first organisms that appeared about 3.5 billions years ago were prokaryotes. They developed primitive forms of photosynthesis without oxygen as a waste. The first photosynthesizing prokaryotes appeared about 2.7 billion years ago which led to the changes in Earth atmosphere and consequently organisms dependant on oxygen could arise.</span>
No animals get nitrogen from eating plants or other animals that contain nitrogen and plants get nitrogen from the soil by absorption through their roots as amino acids, nitrate ions, nitrite ions, or ammonium ions.
1. Regulation of glucose blood levels is an example of negative feedback mechanism.
Negative feedback mechanism is a control mechanism involved in homeostasis maintain, in this case maintenance of glucose blood levels in normal range.
Negative feedback mechanism contains sensory system that detects the changes, control system that responds to change and activates mechanisms of effector system that reverse the changes in order to restore conditions to their normal levels.
• Pancreatic cells-sensors
• Insulin-control system
• Body cells- effector cells
2. Blood glucose levels change throughout the day because of the food consumption, but in healthy individuals levels of glucose are successfully regulated via the mechanism of hormones such as insulin and glucagon in a process called glucose blood regulation.
This tight regulation of pancreatic hormones is referred to as glucose homeostasis. Insulin lowers blood sugar and glucagon raises it.
3. If the beta cells are destroyed by an autoimmune disease (immune system attacks its own cells), there would be no insulin release, and consequently, the glucose blood levels would be increased.
Diabetes type I is a metabolic disorder caused by the destruction of insulin-producing pancreatic beta cells.
