Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
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Answer:
Explanation:
An organism can't chose to have a mutation because it depends on the way they are born. If they are missing some type of mineral, (Protein, Vitamin, etc.) they will have a mutation because they're missing that. Well, something similar to a mutation is natural selection and genes. For example, there are some short and tall giraffes. The tall ones can reach the tree and the short ones can't so they die and the tall ones can reproduce and their offspring will inherit that trait
Answer:
This is an example of "Disruptive selection".
Explanation:
<em>Disruptive selection</em> occurs when <em>selective pressure</em> <em>favor homozygous</em>. In equilibrium, <em>the two alleles might be present or one of them might be lost</em>. If an environment has two extremes, then in these environments, both alleles are presented in homozygous.
The disruptive selection causes an <em>increase</em> in the two types of <em>extreme phenotypes over the intermediate forms</em>. Limits between one extreme and the other are frequently very sharped. Individuals belonging to one phenotype can not live in the same area as individuals belonging to the other phenotype, due to the traits differences between them, competition, or predation.
Populations show two favored extreme phenotypes and a few individuals in the middle. Individuals who survive best are the ones who have traits on the <u>extremes forms</u>. Individuals in <u>the middle</u> are not successful at survival or reproduction.
<em>Color</em> is very important when it comes to <em>camouflage</em>. Dark green caterpillars that live in dark foliage and light green caterpillars that live in light foliage can <em>hide from predators</em> more effectively and will live the longest. Intermediate colored green caterpillars that don't camouflage or blend into either will be eaten more quickly.
Answer:
Nephrons in birds, mammals, and reptiles are all extremely similar, more so than other structures in the bodies of different species, solidifying the relatedness through similarity.
Explanation:
Birds can be said to have "mammal-like" nephrons from the number of loops and overall structure of their kidneys, which, although they look very different, serve the same purpose and do it in largely the same way. Reptiles also have mammal-like nephrons, and it can be assumed that this evolutionary trait was kept because the specific structure of the nephrons is generally the most efficient.
Edema is a condition of abnormally large fluid volume in the circulatory system or in tissues between the body's cells(interstitial <span>spaces).</span>