<span> </span><span>Bean roots will have no nodules if there are no compatible Rhizobium bacteria in the soil. The nodule is a symbiotic relationship between the plant and the bacteria. Nitrogen fixing root nodules are pinkish in color. Green root nodules indicate actively reproducing bacteria that are not fixing nitrogen. The number of nodules depends on the amount of innoculant (Rhizobium) available in the soil. Look at the photos and make an estimate like x nodules per y linear inches of root. Hope this helps.
The edible part of the radish root functions as a food storage organ. The string parts of the radish root function as normal roots absorbing water and dissolved nutrients. Bean roots have no modifications for food storage. Radish roots don't have nodules. The radish root is a "tap root". The bean root is a "fiberous root"
Beans don't grown faster than radishes because of the root nodules. Bean seeds are large. Radish seeds are small. The energy stored in the bean cotyledons helps the seedling get a fast start. The radish gets a slower start from less stored energy.</span>
Answer:
In photosynthesis it occurs on the thylakoid membranes.
Explanation:
It is where the light independent reaction occurs.
Answer:
Your answers are probably estrogen and oxytocin.
Answer:
1/2
Explanation:
Let's assume that the allele "p" is responsible for PKU in the homozygous state. According to the given information, both the parents are unaffected by the disorder but have an affected child. This means that both the parents carry at least one copy of the recessive allele responsible for the disease. Therefore, the genotype of each of the parents is Pp. A cross between Pp and Pp would produce progeny in following ratio=
Pp x Pp = 1/4 PP: 1/2/ Pp: 1/4 pp
Therefore, the probability that their next child will carry just one recessive allele is 1/2.
Answer: DF508 mutation. A Genetic, Hereditary, Autosomal and Recessive Mutation.
Explanation:
Cystic fibrosis (CF) is a recessive autosomal lethal disease, it is most common on Caucasoid populations. Its diagnosis is suggested by the clinical features of chronic obstructive pulmonary disease, persistent pulmonary colonization (particularly with mucoid Pseudomonas strains), meconium ileus, pancreatic insufficiency with or familiarity history of the disease. The FC gene is large, with about 250 Kb of genomic DNA, 27 exons representing about 5% of genomic DNA; encodes a 6.5 kb transcribed mRNA. This mRNA is transcribed into a protein of 1480 amino acid called CFTR (Regulator Transmembrane Conductance Cystic Fibrosis). When a three-base pair deletion, adenosine-thymine-thymine (ATT) identified in the CFTR gene, exon 10, it results in the loss of a single amino acid phenylalanine at position 508 of the protein. This mutation is called DF508; “D” stands for deletion and “F” for phenylalanine amino acid.
