It’s corresponding molecule that or bonds to. I think it is a substrate.
A mechanism that is effective in maintaining a normal glomerular blood pressure only if the systemic mean arterial pressure remains between 80 and 180 mm hg is called renal autoregulation.
<h3>What is renal autoregulation?</h3>
- Autoregulation is the inner characteristic of blood vessels present in end organs, like heart, kidney, and mind, by which they dilate or constrict in response to pressure changes, thus help to keep the blood flow generally steady.
- Usually our body tries to regulate our blood pressure in range of 50 to 150 mm Hg.
- Regulation of renal blood flow and glomerular blood pressure in kidneys is called renal autoregulation.
- There are 3 mechanisms of renal blood flow namely myogenic response (MR), tubuloglomerular feedback (TGF) and third mechanism that is independent of TGF but slower than MR.
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Answer:
The microbes thriving in acidic environments are termed as acidophiles, and these range from eukaryotes to bacteria and archaea, which are mainly found in diverse acidic surroundings like sulfuric geysers and pools, in the human stomach, and in the regions that get polluted by acid mine drainage.
The mentioned case is not entirely correct as the protons found in the acidic surroundings are not utilized for the generation of ATP as they are not originating from within the cell. In order to sustain their internal acidic pH, the acidophiles exhibit adaptations like the presence of the negatively charged proteins on the surfaces of their membranes so that they can prevent deterioration due to acidic surroundings.
Lactase refers to an enzyme that can dissociate lactose sugar into glucose and galactose. This enzyme plays an important role in the digestion of milk that comprises high lactose sugar. In case, if a mutation occurs in the gene codon of lactase, then two possibilities can take place. These are as follows:
1. Loss of mutation: In this case, the person becomes lactose intolerant because of mutation in lactase gene codon. Due to this, there is low mRNA expression and thus low production of the enzyme lactase. This is also known as lactase non-persistent phenotype.
2. Gain in mutation: In this case, the person gain an increase in mRNA expression of the lactase gene, thus more production of lactase takes place than usual. This kind of individual exhibits lactase persistent phenotype.
Answer:
The probability that the child will have type blood B equals <u>3/16</u>.
Explanation:
<u>Available data:</u>
- Individuals with the rare Bombay blood phenotype lack both the A and B antigens in individuals and/or are of hh genotype.
- Cross between two parents that are both of I A I B Hh genotype
Cross: IAIB Hh x IAIB Hh
Gametes) IAH, IAh, IBH, IBh
IAH, IAh, IBH, IBh
Punnett square) IAH IAh IBH IBh
IAH IAIAHH IAIAHh IAIBHH IAIBHh
IAh IAIAHh IAIAhh IAIBHh IAIBhh
IBH IAIBHH IAIBHh IBIBHH IBIBHh
IBh IAIBHh IAIBhh IBIBHh IBIBhh
F1) Genotype
- 1/16 IAIA HH
- 2/16 IAIAHh
- 1/16 IAIAhh
- 2/16 IAIBHH
- 4/16 IAIBHh
- 2/16 IAIBhh
- 1/16 IBIBHH
- 2/16 IBIBHh
- 1/16 IBIBhh
Phenotype
- 3/16 Blood type A
- 6/16 Blood type AB
- 3/16 Blood type B
- 3/16 Blood type 0
