Answer:
It made it possible for people to discover and learn about cells.
Explanation:
The cell is the basic unit of structure and function of all living beings. A cell can also be defined as a morphological, functional, and reproductive unit of all living beings.
Every living organism is made up of one or more cells. All cells are created from an existing cell. A cell is the smallest unit that has all the characteristics of life. A set of cells of similar or the same appearance, embryonic origin, and function is called tissue.
The science that studies the cell is called cytology. There are organic and inorganic compounds in the cell. Of the inorganic compounds, water and salts are the most common. Organic compounds in the cell contain carbohydrates, fats, and proteins.
Answer:
c
Explanation:
Gas exchange. Gas exchange occurs as a result of respiration, when carbon dioxide is excreted and oxygen taken up, and photosynthesis, when oxygen is excreted and carbon dioxide is taken up. The rate of gas exchange is affected by: the area available for diffusion.
Answer:
Explanation:
when youre feelinsgs subside and shadows still remain
gons and ruse
They changed things up because its mixed or from up further in the family line. like your parents could have green eyes and you could get blue. it shoukd of came from your grandparents or other blood related relatives.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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