It’s not an autosomal disease at all
Answer:
It decreases the levels of cAMP in the cell, repressing transcription from the lac operon.
Explanation:
When glucose is absent, cAMP serves as coactivator binds to CRP, the catabolite gene activator protein. The CRP-cAMP complex binds to the site near the lac promoter and stimulates the expression of the operon by RNA polymerase many folds.
Catabolite repression refers to inhibition of the synthesis of enzymes of lactose catabolism when glucose is present as an energy source. In the presence of glucose, synthesis of cAMP is inhibited resulting in its lower cellular concentration. The lower cAMP levels do not allow the binding of cAMP and CRP. The result is reduced expressed of lac operon.
<span>well this is an interesting question and i would say it may depend on what type of cancer cells you are growing and what type of "normal" cells your growing. One possibility is that cell fusion events may occur between your cancer cells and normal cells, thus creating a few options 1 - making the normal cell cancerous, 2 - making the cancer cell that fused with the normal cell not cancerous anymore. 3 - either way the fused cell will have a different genotype and hence be a different cell.</span>
Answer:
Dichotomous keys can only be used to classify organisms based on their observable traits.
Explanation:
I got it wrong on connexus so i hope you dont make the same mistake i did ;-;
