Grade aa eggs are nearly perfect
He demonstrated that SCD and sickle cell trait were due to the presence of abnormal 8-globin polypeptides in red blood cells. He demonstrated that the electrophhoretic mobility of B-globin from patients with SCD was different from that of healthy individuals. He demonstrated that both parents of multiple patients with SCD had low levels of sickled red blood cells. He hypothesized that SCD was a recessive trait and that the parents of patients with SCD would be heterozygous carriers. He demonstrated that the difference between B-globin polypeptides in individuals who were healthy and those with SCD is an amino acid substitution. He performed a peptide fingerprint analysis on B-globin from individuals with 84 84 and 89 88, which identified the segment of B-globin that was changed by the BS mutation. James Neel Linus Pauling Vernon Ingram
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Answer: C. There is strike-slip fault between the Pacific and the Northern American plates.
Explanation:
Answer:
The protein likely travels through a common lumen shared by thylakoid membranes and grana, and cannot easily diffuse through the thylakoid membrane.
Explanation:
There is a lot of scientific research in which a specific molecule can be labeled with some fluorescent marker (usually carbon 14). This type of marking allows the researcher to make observations about the movement of these molecules, as you can see in the question above. About the research shown in the question, the researcher realized that the protein labeled with the fluorescent marker moved between the grana and was always in the lumen, so she can conclude that the selocomovement protein moved through the lumen that is shared between the tilacoid membranes and the grana.
One of the plants in this grouping are chrysanthemums.
