Systemic lupus erythematosus (SLE) is a rheumatic disease attributed to autoimmune mechanisms.
Systemic lupus erythematosus (SLE, lupus) is characterized by a global loss of self-tolerance with activation of autoreactive T and B cells, leading to the production of pathogen autoantibodies and tissue damage.
Innate immune mechanisms are required for aberrant adaptive immune responses in SLE. Recent advances in basic and clinical biology have shed new light on the disease mechanisms of lupus. This review article describes recent studies that provide valuable insights into disease-specific therapeutic targets.
Systemic lupus erythematosus (SLE or lupus) is a systemic autoimmune disease with inflammation of multiple organs. SLE is characterized by the production of pathogenic autoantibodies against nucleic acids and their binding proteins, reflecting a global loss of self-tolerance.
Loss of tolerance with subsequent immune dysregulation is the result of genetic factors in the context of environmental triggers and stochastic events, with recent studies implicating more than 30 genetic loci in disease pathogenesis.
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Answer:
Scoliosis can lead to osteoarthritis and degenerative changes in the spine, hips and knees. If a case is severe enough, internal organs may also be compromised or severely damaged.
Explanation:
The cause of physiological scoliosis is the human gait, which forces the spine into a threefold curve, alternately changing at every step. However, the innate normal asymmetries of the human body eventually establish permanent lateral curves of the spine, showing marked individual variations within the general law.
Answer:
pubic hair and underarm hair
Explanation:
Because both sexes get these trough puberty
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