Answer:
desert
Explanation:
they both have plants and animals that need special adaptations to survive there
Answer:
Nile crocodile
Explanation:
The Egyptian plover is a small bird with a very unusual friend in the animal kingdom. This bird actually has a symbiotic relationship with the Nile crocodile. It may seem very odd relationship, but it is one that works excellently for both sides. The crocodiles have sharp set of teeth, but they have spaces between them and can not move their jaws sideways. Because of this, very often there's meat stuck between the teeth of the crocodiles, and this is when the Egyptian plover comes on the scene. This little bird actually gets into the open mouth of the crocodiles, picking up the pieces of meat that have stuck between their teeth, and then continuing with its mission. The crocodiles do not try to eat this bird, but instead they stand still while it does its job. The relationship is beneficial for both sides, as the crocodiles have their own personal ''dentist'', while the plover gets free food.
Winds will blow eat to west
Scientists consider the INTERPHASE the resting phase of the cell cycle because they can not see a visible change during this phase.
The interphase refers to the interval in the cell cycle between two cell divisions. During this stage, the individual chromosomes in the cell can not be distinguished. Although scientist can not see any outward visible change during this phase, but it is actually the time when the DNA inside the nucleus is replicated.
<h2>CRISPR/Cas9</h2>
Explanation:
CRISPR can be used to reintroduce dystrophin back into the KO mouse
- CRISPR stands for Clustered Regularly Interspaced Short Palindromic Repeats and is used to for gene editing
- CRISPR/Cas-mediated genome editing has been shown to permanently correct DMD mutations and restore dystrophin function in mouse models
- Germline editing by injecting zygotes with CRISPR/Cas9 editing component was first done in mdx mice by correcting the mutated exon 23
- Postnatal editing of mdx mice was then achieved using recombinant adeno-associated virus to deliver CRISPR/Cas9 genome editing components and correct the dystrophin gene by skipping or deleting the mutated exon 23 in vivo
- Germline and postnatal CRISPR/Cas9 editing approaches both successfully restored dystrophin function in the mice and same technique can be used for KO mouse model
