The answer is perhaps neurons ......
The sarcomere will have uncontrolled muscle contractions.
Troponin blocks myosin-binding sites in the tropomyosin of the actin. If the troponin is damages, the myosin-binding sites will be exposed and myosin heads (of the myosin fiber) will be able to form cross-bridges with the actin even when not required.
Explanation:
This means the sarcomere will contract even when not required or expected hence resulting in symptoms like muscle spasms. Usually, troponin molecules exposes the myosin-binding sites on the tropomyosin upon binding of Ca²⁺ ions. These Ca²⁺ are usually released by the sarcoplasmic reticulum upon impulse from the inverted nerve.
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