The answer is Ingested corrosives may be diluted by drinking 4–6 oz (113.4–170.1 ml) of water or milk. If vomiting is persistent, do no attempt to administer additional fluids. Avoid neutralization therapies as the resultant exothermic reaction may cause additional tissue injury.
Explanation :
- Strong acids, corrosive agents, and highly reactive oxidants can cause large-scale destruction of living cells through direct chemical reactions. Sometimes referred to as necrotic cell death, this non-specific effect is induced by concentrated solutions of caustic and caustic chemicals that cause indiscriminate cell destruction.
- A generalized overwhelming effect of this type is no different from those resulting from "burning" tissue. These chemically induced injuries are commonly referred to as "chemical burns."
- Such effects are produced not only by strong acids or bases in harmful concentrations, but also by exposure to concentrated solutions of organic solvents such as ether, chloroform or carbon tetrachloride. The intensity of such non-specific toxicity is directly related to the concentrations of the chemical agents when in contact with the target tissues.
- Widespread cell destruction can be caused by any chemical that is sufficiently soluble in tissue fluids to access cells at high concentrations. The effects of these chemicals in higher organisms are generally limited to easily accessible tissues such as the skin, eyes, mouth, nasal mucosa, and respiratory tract.
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Answer:
a. sighing.
b. concentrates on feelings of warmth and heaviness
Explanation:
Positioning. Multiple positions may be used to accomplish a digital rectal examination. The easiest for the examiner is to have the patient tuck the knees up to the chest, either in the dorsal lithotomy position or the lateral recumbent position.
The acidic urinary pH increases the formation of uric acid or cysteine crystals. Therefore, kidney stones are very much influenced by urinary pH. More acidic urine and a higher frequency of uric acid stones have been associated with patients with inadequate ammonium excretion of metabolic syndrome.
Kidney stone disease results from deficiencies in urinary acidification, which result in the excretion of improperly alkaline or acidic urines, respectively. Patients with uncommon mendelian kidney stone variants have mutations in several enzymes, transporters, receptors, or channels. Numerous of these alterations either result in an increase in the excretion of chemicals that can crystallise or stone, a change in the composition of the urine that promotes crystal formation, or both.
Kidney stones can occur as a result of a variety of variables
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