Answer:
A distinct phase during the stride cycle where the runner's weight is carried by the entire foot.
Explanation:
Answer:
The correct answer is: B, C, D,F, G, H.
Explanation:
Aneuploidy is a defect on the number of chromosomes a cell has. Human cells have 46 chromosomes, an additional chromosome (47) or a missing one (45) is an aneuploidy. Aneuploidy is usually due to a defect during cell division (chromosomes don’t separate). The following disease are examples of aneuploidy:
b. Down's syndrome (trisomy of chromosome 21)
c. Edward's syndrome (trisomy of chromosome 18)
d. Jacob's syndrome (a male with an extra Y chromosome)
f. Turner's syndrome (a female missing a X chromosome)
g. Klinefelter's syndrome (a male with an extra X chromosome)
h. Patau's syndrome (trisomy of chromosome 13)
Answer:
If your amputated finger can be saved, you may need microsurgery. Your doctor will sew together the nerves, blood vessels, and tendons inside your finger. This helps to keep your finger alive and heal well after it's reattached.
Explanation:
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Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
Answer:
dose and temperature of treated volume
