Answer:
I'm guessing it would be HIPAA
Explanation:
Answer:
(B). inhibiting HMG-CoA reductase.
Explanation:
Lovastatin is a drug, which is used to reduce risk for cardiovascular diseases and to reduce level of cholesterol in blood.
It works by inhibiting HMG-CoA reductase (3-hydroxy-3-methylglutaryl-coenzyme A reductase) enzyme, required for the formation of mevalonate from HMG-CoA.
Mevalonate is building block for the biosynthesis of cholesterol. Hence, lovastatin inhibits cholesterol production by inhibiting synthesis of mevalonate.
Thus, the correct answer is option (B).
The information collected by the nurse who is admitting a patient with chest pain suggests that the pain is caused by an acute myocardial infarction (AMI) the pain has persisted longer than 30 minutes.
- A blood clot or other obstruction of the flow of blood to the heart muscle causes a heart attack. Angina. The term "angina" refers to chest pain brought on by inadequate cardiac blood supply. This is frequently brought on by the development of thick plaques on the inner walls of the arteries that supply the heart with blood.
- AMI, more commonly known as a heart attack in layman's words, is most frequently brought on by a reduction or cessation of blood supply to a section of the heart, which results in the necrosis of heart muscle. Usually, a blood clot in the artery that supplies that region of the heart muscle causes this.
To learn more about chest pain.
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Answer:
<u>Ethanol prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u>
Explanation:
Ethylene glycol is an organic compound usually used in antifreeze solutions, solvents and cleaners.
It is abused during self-destruction and accidental intakes.
<u>In the body, ethylene glycol is acted upon by alcohol dehydrogenase and is converted into glycolate and oxalate.</u>
Glycolate and oxalate are both nephrotoxic/ kidney damaging substances. Oxalate precipitates calcium oxalate stones in the kidney. Ethylene glycol poisoning also causes high anion gap metabolic acidosis.
In order to prevent ethylene glycol poisoning, the patient is infused with ethanol, ethanol <u>prevents toxicity by competing with Ethylene glycol for metabolism by alcohol dehydrogenase.</u> In this way, ethylene glycol is not metabolized and the formation of nephrotoxic substances is prevented. Alcohol dehydrogenase instead reacts with ethanol to form acetaldehyde.
