Answers:
a) carcinogenic
b) anti-carcinogenic
c) carcinogenic
d) carcinogenic
e) carcinogenic
f) anti-carcinogenic
g) anti-carcinogenic
h) anti-carcinogenic
Explanation:
Cyclins are proteins that regulate the progression through the cell cycle, i.e., the transition of G1 to S phase. It is well known that high cyclin expression may lead to cell proliferation states, which is closely associated with cancer progression. Moreover, the blockage of cyclins may have an anti-carcinogenic effect by inhibiting the progression through the cell cycle. MAP kinases are serine/threonine kinases that regulate the progression through the cell cycle by phosphorylating a variety of substrates during cell proliferation. In consequence, phosphatases that inactivate MAPK kinases (i.e., by dephosphorylation) may have an anticarcinogenic effect. The p53 is a tumor suppressor protein involved in diverse cellular processes including DNA repair, cycle arrest and programmed cell death. This protein (p53) is activated by phosphorylation at target residues and phosphatases inactivate it, thereby the blockage of its degradation may have an anticarcinogenic effect. Oncogene activation (i.e., the expression of oncogenes), may alter diverse cellular processes including DNA replication, and thereby may lead to cancer development. The G-protein α subunit is a GTPase that hydrolyses GTP and thus has a major role in controlling the kinetics of the G-protein signaling cascade. Platelet-derived growth factor receptors (PDGFR) are kinase receptors that play roles in regulating cellular differentiation, cell proliferation and cell growth. PDGFR receptors are present on the surface of normal cells, however, it has been shown that mutations of the PDGFR genes that lead to their high expression lead to uncontrolled cell growth and consequently cause cancer (i.e., by increasing PDGF signaling).
C. Anaphase
Anaphase is the briefest stage of mitosis. The centromeres begin to separate, being pulled to the poles by the spindle fibers. As the centromeres seperate the chromatids split.
The reduced potential causes hundreds of <u>voltage-gated sodium</u> channels to open on that part of the cell membrane. The depolarization of the cell causes more of <u>voltage-gated sodium </u>channels to open in adjacent parts of the cell membrane. This begins the wave of of <u>depolarization</u> moving down the axon. Depolarization begins at the <u>axon hillock.</u>
Explanation:
When there is no neuron signaling it becomes polarized, termed as resting membrane potential (RMP) at a threshold voltage (around -55 mV), due to the action of the sodium-potassium pump and the potassium leak channels.
When a change in the RMP occurs, depolarization takes place which causes the voltage-gated sodium channels to open and sodium ions rush into the nerve cell which in turn will increase the voltage threshold to nearly around +40 mV and also charges the neuron positive. This depolarization moves down the axon. This increase in threshold stops the sodium influx and opens the potassium channels to rush the potassium out of the cell.
All these actions decrease the membrane potential leading to a wave of depolarization and going back to resting state. Depolarization begins depending upon the potential gradient at the axon hillock.
Answer:
The answers is D option why because when he jumps and goes down with a force and opens parachute so lesser force with his acceleration
