Answer:
The correct answer would be the harmless bacteria had been transformed.
Griffith used two different strains of the bacteria <em>Streptococcus pneumoniae - </em>type-III-S or smooth strain and type II-R or rough strain.
Smooth strain had protective covering around itself (protect itself from hosts's immune system) and was able to kill the mice.
Rough strain did not have any protective covering around itself and thus could be easily removed by the immune system Hence, it was not able to kill the mice.
In addition, heat killed smooth strain was also not able to kill the mice. However, when remains of it was added with rough strain then the blend was able to kill the mice.
Lastly, he was able to isolate living bacteria of both the strains.
He concluded that non-lethal type II-R strain was transformed into lethal type II-S strain by "transforming principle" (which we know today as DNA) that was supposed to be the part of dead III-S strain bacteria.
They are joined together by a common centromere.
Portals of entry are the sites where micro-organisms can enter a host and reproduce, causing diseases or infections. There are various portals of entry in the human body. This can include the skin, our mucous membranes, and even our respiratory and gastrointestinal tracts. Portals of exit are where these micro-organisms leave and spread to other individuals. Portals of exit include coughing, sneezing, and some of our bodily fluids.
Answer:
grasshopper
The organisms that eat the producers are the primary consumers. ... The primary consumers are herbivores (vegetarians
Answer:
Pseudopodia are temporary and cytoplasm-filled parts of the cell membrane that are able to change their form in order to move.
Explanation:
Pseudopodia are temporary and cytoplasm-filled parts of the cell membrane that are able to change their form in order to move. They are used in some eukaryotic cells to move around or to eat. Most cells that do this are called amoeboids. The amoeba is a common example. ... Pseudopods can also capture prey by phagocytosis.
