Answer:
c
Explanation:
sperm
What are produced in both the male and female reproductive systems in humans?
Answer:
Molecular chaperons in the cells helps in protein folding. These are the group of proteins that have functional similarity and they also assist protein folding.
They have the ability to prevent the non specific binding and aggregation by the binding of the non native proteins.
Molecular chaperons helps in recognizing the hydrophobic surfaces of the unfolded proteins because they themselves are hydrophobic in nature and will combine to the hydrophic binding and bonding.
This helps in guiding the protein to folding.
This statement is mostly false. While all organisms do share basic needs to sustain life, there is a wide variety of strategies employed to meet these needs. For example, while most plants receive nutrients from soil, some live in soils which do not fully meet their needs. Such plants sometimes kill and use animals in order to meet these needs (i.e. venus flytrap) or partner with bacteria that produce some of the nutrients they need (i.e legumes).
Answer:they won't for a pair
Explanation:Basically it all depends on process called complementary base pairing, the structure of thymine and adenine is such that it forms two hydrogen bonds and therefore pair up
2.guanine and cytosine have such structure that they form three hydrogen bonds and pair up
So Adenine complements only thymine and guanine complements cytosine and there aren't any chances of guanine pairing up with thymine.
Temperature affects spermatogenesis, which functions best at body temperatures just a little lower than those.
<h3>Abstract:</h3>
To keep testicular temperatures below those of the body core, adequate thermoregulation is essential. The process of mammalian spermatogenesis and the resulting spermatozoa are negatively impacted by elevated testicular temperature. Therefore, sperm quality can be affected and the likelihood of infertility is increased by thermoregulatory dysfunction resulting in heat stress. This article reviews a variety of internal and external factors that may lead to testicular heat stress. We go into more detail on how heat stress affects the spermatogenesis process, the resulting epididymal spermatozoa, germ cells, and the alterations that result in the testis.
We also go over the chemical reactions of germ cells to heat exposure and potential processes, such as apoptosis, DNA damage, and autophagy, that could lead to heat-induced germ cell damage. Further explanation is provided for the intrinsic and extrinsic processes involved in the complex mechanism of germ cell death. These intricate apoptotic pathways ultimately result in the demise of germ cells.
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