Answer:
700
Explanation:
If from the question the price per unit of fertilizer, p(x) = 300 - 0.1x
The cost of x units of fertilizer = C(x) = 15000 + 125x + 0.025x^2
And we know that Profit = Revenue - Cost
Revenue for x units of fertilizer R(X) = x*p(x)
= 300x - 0.1x^2
Hence Profit: P(x) = R(x) - C(x)
= [300x - 0.1x^2] - [15000 + 125x + 0.025x^2]
= -0.125x^2 + 175x - 15000
To determine critical values
P'(x) = -0.25x + 175 = 0
Thus x = 175/0.25
x = 700
To maximize profit, the manufactures must produce 700 units of fertilizers
Answer:
The sequence of nucleotides, coded in triplets (codons) along the mRNA, that determines the sequence of amino acids in protein synthesis. The DNA sequence of a gene can be used to predict the mRNA sequence, and the genetic code can in turn be used to predict the amino acid sequence.
Explanation:
Answer:
Over time the beetle population will adapt as a result of natural selection because the beetles "desired" by the birds will be eaten and their population will dimish as a result. The beetles that are not eaten by the birds will have a chance to allow their population to grow because a. they aren't being eaten and b. they have more space as the other colored beetle's population is shrinking. Causing the eaten species to become less prominant and the not eaten beetle's population to grow and take over in place of the other beetles.
Based on that scenario, a faulty reasoning that the student's research advisor most likely point out is : The rock that makes up the fossils replaced the organisms tissue and genetic material,
Because of this , their genomes can't be extracted.
hope this helps
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
To learn more about protein-protein interaction visit:
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