<span>In liver, the most intensively studied transmembrane and intracellular signal transduction pathways are the Janus kinase signal transduction pathway, the mitogen-activated protein kinases signal transduction pathway, the transforming growth factor β signal transduction pathway, the tumor necrosis factor α signal transduction pathway and the recently discovered sphingolipid signal transduction pathway. All of them are activated by many different cytokines and growth factors. They regulate specific cell mechanisms such as hepatocytes proliferation, growth, differentiation, adhesion, apoptosis, and synthesis and degradation of the extracellular matrix. The replication cycle of hepatitis C virus (HCV) is intracellular and requires signal </span>
Processes that are most important to human life are oxygen, required for cellular respiration, and glucose, a form of sugar that releases energy during cellular respiration.
Answer:
Carbon monoxide and antimycin are classified as electron transport inhibitors, FCCP as uncoupling agent, oligomycin as ATP synthase inhibitors, and bongkrekic acid as transport inhibitor.
Explanation:
The electron transport inhibitors refers to the substances, which get combine with distinct constituents of the ETC and prevent the function of the carrier. These substances binds with the carrier and prevent its transformation, which eventually results in halting of the process. The most commonly known electron transport inhibitors are rotenone, carbon monoxide, antimycin, etc.
A molecule that prevents oxidative phosphorylation in mitochondria and prokaryotes, or photo-phosphorylation in cyanobacteria and chloroplasts by inhibiting the production of ATP is known as ATP synthase inhibitors. The most commonly known ATP synthase inhibitor is oligomycin.
Similarly to oligomycin, FCCP or trifluorocarbonylcyanide phenylhydrazone is a potent uncoupler or uncoupling agent that prevents the synthesis of ATP by enhancing membrane proton permeability.
Bongkrekic acid refers to a highly toxic transport inhibitor that prevents the ADP/ATP translocase by combining with the inward facing site of ATP-ADP translocase.
