Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.
It's called t<span>he trachea.</span>
It measures the total rate at which photosynthesis or chemosynthesis happens in a system.
1-metaphase, since an ecuatorial plate is formed
2.prophase since the nuclear envelope is starting to dissapear
3.Telophase since new nucleae started to form around each separated genetic material
4.Interphase
5.Interphase
6.Interphase
7.anaphase since genetic material(chromsomes) is pulled to the poles of the cell
8.Interphase
9.Anaphase
10.Interphase
11.metaphase
12.telophase
13.Interphase
14.Prophase
15.interphase
16.interphase
17.metaphase
18.interphase
