Answer:
159.808 g/mol
Explanation:
Br itself is 79.904 g/mol. So multiply it by two.
Answer:
c. Increased reabsorption of phosphate in the kidneys
d. Increased deposition of calcium into the bones.
Explanation:
Hyperphosphatemia is a condition that is expressed particularly in people with a kidney dysfunction. It comprises the kidneys, which do not excrete enough phosphate from the body as they reabsorbe it and thus leading to increased phosphate levels.
Also, phosphate binds calcium with high affinity, provoking acute hypocalcemia (decreased levels of calcium). In Hyperphosphatemia, calcium is being deposited mostly in the bone but also in the extraskeletal tissue.
Answer:
(1) glycerophospholipids ⟶ (C) lipids with phosphate-containing head groups
(2) cerebrosides ⟶ (D) fatty acid linked through an amide bond to the sphingosine C(2)-amine
(3) gangliosides ⟶ (B) anionic sphingolipids containing one or more sialic acid residues
(4) sphingolipids ⟶ (A) built on sphingosine
Explanation:
1) Choline (Fig. 1) is a glycerophospholipid. It is a glycerol-based lipid with a phosphate-containing head group.
(2) Galactosylceramide (Fig. 2) is a cerebroside. It contains a fatty acid linked through an amide bond to the sphingosine C(2)-amine
(3) Gangliosides are glycosphingolipids that contain sialic acid. GQ1b (Fig. 3) is one of the most abundant gangliosides in the human brain. The carboxyl group of the sialic acid is in the ionic form.
(4) Sphingomyelin (Fig. 4) is a sphingolipid. It is based on sphingosine, with a phosphocholine head and a fatty acid chain.
Answer:
Carbon dioxide + water - hy cose + oxygen
Explanation:
<span>This indicates that energy consumption could have fluctuated. </span>