Answer:
Electron transfer to from cytochrome c to molecular Oxygen in the process of oxidative phosphorylation
Explanation:
Cytochrome c is a protein which is involved in the electron transport chain for the production of ATP molecules during then process of respiration. It a soluble protein found in the intermembrane space of the mitochondria. It receives electrons from ubiquinone at Complex III of the electron transport chain and transfers this electron to molecular oxygen through its interaction with complex IV or cytochrome c oxidase, reducing molecular oxygen to water.
If the interaction of cytochrome c with cytochrome c oxidase is inhibited, the process of elctron transfer to oxygen will be inhibited and, so ATP synthesis will cease.
Ultimately, respiration will be inhibited resulting in death of the organism. For example, cyanide inhibits cytochrome c oxidase resulting in death of the organism poisoned with cyanide.
Answer:
B.
Explanation:
potential energy changes into kinetic energy
.Oxygen free environments
.Concentrated salt water and hot
. Acidic water of sulfur springs
Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
