Answer:
The processing power of the mammalian brain is derived from the tremendous interconnectivity of its neurons. An individual neuron can have several thousand synaptic connections. While these associations yield computational power, it is the modification of these synapses that gives rise to the brain's capacity to learn, remember and even recover function after injury. Inter-connectivity and plasticity come at the price of increased complexity as small groups of synapses are strengthened and weakened independently of one another (Fig. 1). When one considers that new protein synthesis is required for the long-term maintenance of these changes, the delivery of new proteins to the synapses where they are needed poses an interesting problem (Fig. 1). Traditionally, it has been thought that the new proteins are synthesized in the cell body of the neuron and then shipped to where they are needed. Delivering proteins from the cell body to the modified synapses, but not the unmodified ones, is a difficult task. Recent studies suggest a simpler solution: dendrites themselves are capable of synthesizing proteins. Thus, proteins could be produced locally, at or near the synapses where they are needed. This is an elegant way to achieve the synapse specific delivery of newly synthesized proteins.
Explanation:
Answer: c. the physical movement of air into and out of the lungs.
Explanation:
Pulmonary ventillation is also called as breathing. This is the process which involves the inhalation or physical entry of air inside the lungs this process is called as inspiration. And the flow out of air out of the lungs is called as exhalation. The flow of air creates a pressure difference between the atmosphere and the lungs.
Answer:
Superior Vena Cava -> Right Atrium -> Right Ventricle -> Pulmonary Artery -> Lungs -> Left Atrium -> Left Ventricle -> Aorta -> Rest of Body
The pathophysiology instructor who is teaching nursing students about chronic pancreatitis should stress upon its complications like <u>pancreatic necrosis,</u> fluid and electrolyte imbalance or disturbances, and septic shock.
Explanation:
The pathophysiology of chronic pancreatitis includes destruction of the fibrous pancreatic secretory parenchyma with intra and extracellular loss, obstruction of ducts and transport of pancreatic secretions, and triggering of enzymatic activities.
These conditions lead to autodigestion and histological destruction of pancreatic tissues leading to necrosis and complete organ damage.
Pancreatic necrosis can result in hemorrhagic or septic shock which can further lead to multiple organ failure. Hence, pancreatic necrosis is a major complication leading to morbidity and mortality among pancreatitis patients.
The structural damages lead to dilatation and malfunctioning of the pancreatic ducts which results in fluid and electrolyte imbalances.
Someone laid low with osteomalacia is liable to institutional <u>bone fractures</u>.
The maximum commonplace reason for osteomalacia is a loss of diet D. In kids osteomalacia is referred to as rickets humans at risk for developing gentle bones generally possess one or more bone factors associated with low nutrition D stages. these hazardous elements may come from environmental, genetic, and dietary sources.
All and sundry who would not have enough vitamin D are in danger of developing osteomalacia. The best source of nutrition D is sunshine on pores and skin a few humans don't get sufficient sunshine on their pores and skin and this increases their chances.
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