Answer:
Mutations leading to the better attachement of virus to the host cell would allow the virus to spread the infection and leave more progeny. The reproductive fitness of mutant viruses would allow the mutation to spread.
Explanation:
1. The organisms with same resource requirement compete with each other. Viruses are not living things but once inside the host cell, they behave like living things. Rapid multiplication of virus overpopulate the host cells.
2. Genetic variations are added to the viral progeny by random errors during replication of the viral genome. These random variations may impart beneficial trait, harmful feature or remain neutral. If a random mutation in the SARS-CoV2 virus allows it to adhere better to the cells of the respiratory system, the virus would be better able to infect these cells to spread the infection. Once inside the host cells, the mutant virus would leave more progeny. On the other hand, the wild type virus that lack this mutation would have lesser chances of entering the host cells and may or may not replicate.
3. The progeny of the mutant virus would compete better with the non mutant ones due to the presence of same mutation.
4. Differential reproduction and more progeny of the mutant virus would spread the mutation among the virus.
300 B.C theophrastus was considered the "Father of Botanty"
The correct answer is B, 20%
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Answer:
Interrupted mating experiments can determine gene order on the Hfr bacterial chromosome.
Explanation:
Interrupted mating is a technique used in experiments involving microbiology. This experiment was created by geneticists François Jacob and Elie Wollman, in the late 1950s. This technique allows the mapping of the genes of the bacteria involved in the place where the conjugation was interrupted, within monitored and controlled time intervals. In this way, the technique allows showing the transfer mechanisms and the gene transfer itself.
In relation to the question above, we can say that the interrupted mating experiments can determine the order of the genes on the bacterial chromosome Hfr.