Answer:
500
Explanation:
Mutation rate is largely known as changes that occur in the frequency of a particular gene over an extended period of time
So given that;
the mutation rate of a particular gene = 1 × 10⁻⁶
The expected mutations after 500 million sperm are examined will be:
= (1 × 10⁻⁶)×(500,000,000)
= 500
Thus, The expected mutations = 500
Answer:
Elements: H (hydrogen) He (helium) Li (lithium) Be (beryllium) B (boron)
compounds: H2O (water) H2O2 (Hydrogen peroxide) NsHCO3 (baking soda) NaCI (salt)
Element compound or mixture?: 1. Element 2. compound 3. Mixture of elements 4. mixture of compounds 5. mixture of elements and compounds.
Rewrite the sentences:
1. all matter is made up of tiny particles calles atoms and molecules
2. right
3. right
4. compounds are chemically joined together
task 2:
1. i know this is element
2. i know this is compound
3. i know this is mixture
task 3:
In this lesson i have learned that Atoms are in everything and are what makes up most of the stuff on earth. They are tiny molecules that make up me and everything around me. I have also learned that atoms are made up of Elements. Elements are chemical molecules that make up things like the air we breathe. However, when more than one element joins together a compound is formed. Compounds make up more physical things around me such as drinking water and salt for cooking. When many different compounds are joined together a mixture is formed. An example of a mixture can be sand and water.
Explanation:
<u>1</u><u>.</u><u>Antibiotic resistance is a consequence of evolution via natural selection. The antibiotic action is an environmental pressure; those bacteria which have a mutation allowing them to survive will live on to reproduce. They will then pass this trait to their offspring, which will be a fully resistant generation.</u>
<u>2</u><u>.</u><u> </u><u>Bacteria evolve quickly because they grow fast and can share genes. Helpful mutations spread quickly in bacteria.</u>
<u>3</u><u>.</u><u>Taking antibiotics too often or for the wrong reasons can change bacteria so much that antibiotics don't work against them. This is called bacterial resistance or antibiotic resistance</u>
<u>4</u><u>.</u><u> </u><u>Antibiotic resistance leads to higher medical costs, prolonged hospital stays, and increased mortality. The world urgently needs to change the way it prescribes and uses antibiotics.</u>
<u>I</u><u> </u><u>think</u><u> </u><u>so</u><u> </u>
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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Its false because lobsters have swimmerets as well and they dont use them only for swimming
