C) Secure the scene, take photographs, take notes, search for evidence, collect evidence, send evidence to the laboratory for testing.
Answer:
1.What type of reaction is the health care provider concerned about and why?The health care provider concerned about the an IgE mediated allergic reaction. Inthe past, E.O. was given penicillin and had a sign of immune response to the drug.E.O. body has already produce Ig. E. If she uses the penicillin again, it may cause an increased allergic reaction. 2.Explain the role of IgE and mast cells in type I hypersensitivity reactions. Why might E.O. react adversely to the antibiotic with the first use?-The first exposure to an antigen causes B-lymphocytes to produce IgE-The second exposure to the same antigen causes cross links the IgE and mast cell degranulation and release of histamine, serotonin lead to rapid constriction of vessels and causing the hypersensitive reaction. -In the past, E.O.’s body had not produce the IgE immuglobin to penicillin. But at this point, her body is already produce it, and she might has some reaction to the drug. 3.What would you tell E.O.’s parents to look for when they are assessing for a reaction?Itching, angioedema, edema of larynx, uticaria, bronchospasms, vomiting, abdominal cramps, diarrhea
Answer:
Golgi receives a vesicle containing newly synthesized proteins that were sent by the endoplasmatic reticulum. Then it modifies the proteins and sends them where they need to go.
Explanation:
Protein synthesis is initiated in the cytoplasm when mRNA meets a free ribosome, which is the primary structure for protein synthesis. They read the mRNA code and add the correct amino acid using transference RNA to build the protein. The synthesizing protein is driven to the rough endoplasmic reticulum and translocated to the lumen. Once there, the protein suffers a few modifications, one of them is folding to become functional. Once membrane proteins are folded in the interior of the endoplasmic reticulum, they are <u>packaged into vesicles</u> and <u>sent to the Golgi complex</u>, where it occurs the <em>final association of carbohydrates with proteins</em>. The Golgi complex <u>sends proteins to their different destinies</u>. Proteins destined to a certain place are packaged all together in the same vesicle and sent to the target organ. In the case of membrane proteins, they are packaged in vesicles and sent to the cell membrane where they get incrusted.
Question: Describe how a single amino acid substitution causes hemoglobin molecules to stick together. Use what you know about the structure of Hb and HbS, the properties of glutamic acid and valine, and how hydrophobicity causes molecules to behave in water.
Answer:
A Single amino acid must be polar to attract, just like water.
Explanation:
Sickle cell is an genetic illness and it is began by a alteration that arises in the beta sub units of the haemoglobin. Haemoglobin is a tetrameric protein made up of 2 alpha sub units and 2 beta sub units and it is the important part of the blood accountable for oxygen passage. Sickle cell is a illness that consequences from a replacement of a polar amino acid identified as glutamate with a non polar one valine at site six of the beta polypeptide component of haemoglobin. The replacement occurs as a consequence of a alteration in one of the bases in the beta-globin gene from adenine to thymine . As a outcome of this change, the beta polypeptide chains convert sticky in low oxygen circumstances since the valine sticks out of the chain and interrelates with neighboring non-polar amino acids.
The answer is organ systems I believe
