Activation of the Na+/Ca2+ exchanger in the distal tubule is the most likely mechanism of thiazide hypercalcemia
Thiazide-hypercalcemia is a well-known clinical condition. By inhibiting the thiazide-sensitive NaCl transporter in the distal convoluted tubule, which is closely related to calcium transport, thiazides cause an increase in sodium excretion.
Thiazides hypercalcemia decreases urine calcium levels while raising blood calcium levels via promoting calcium reabsorption from the luminal membrane into the interstitium in exchange for sodium. High calcium levels are a well-known adverse effect of thiazide hypercalcemia diuretics including hydrochlorothiazide (Microzide) and chlorthalidone, which are used to treat high blood pressure. By blocking calcium from being discharged in the urine, which can also result in kidney stones, these drugs can boost calcium levels.
To learn more about Thiazides hypercalcemia please visit -
brainly.com/question/10279749
#SPJ4
Let them use models to clean and brush like teeth and make them dirty so they understand how many germs there are and show them a model of how if you don't put deodorant on they will stink and then let them make one up becuase from one teen to another I love it when teachers ask us something or for us to make a decision
Answer:
The correct answer is - Otitis externa.
Explanation:
The given symptoms and examination results most likely presented the idea that she is having Otitis externa. It is a condition that leads to redness and swelling or inflammation of the tube present between the outer ear and eardrum called external ear canal.
This condition is also known as the swimmer's ear due to repeated exposure to water which makes the outer ear canal more prone to inflammation.
Thus, the correct answer is - otitis externa.
