Answer:
i don’t know if it’s a multi question but here are my answer bellow
Claim 1:Cells need glucose to release energy
Claim 3:Cells need oxygen to release energy.
Claim 5:Cells need glucose AND oxygen to release energy
A mechanism that is effective in maintaining a normal glomerular blood pressure only if the systemic mean arterial pressure remains between 80 and 180 mm hg is called renal autoregulation.
<h3>What is renal autoregulation?</h3>
- Autoregulation is the inner characteristic of blood vessels present in end organs, like heart, kidney, and mind, by which they dilate or constrict in response to pressure changes, thus help to keep the blood flow generally steady.
- Usually our body tries to regulate our blood pressure in range of 50 to 150 mm Hg.
- Regulation of renal blood flow and glomerular blood pressure in kidneys is called renal autoregulation.
- There are 3 mechanisms of renal blood flow namely myogenic response (MR), tubuloglomerular feedback (TGF) and third mechanism that is independent of TGF but slower than MR.
Learn more about renal autoregulation here:
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The answer is D, photosynthesis and respiration.
—Evidence—
Oxygen is a byproduct of photosynthesis and, correspondingly, carbon dioxide the byproduct of respiration.
Oxygen gives our cells the ability to break down food in order to get the energy we need to survive. Although other animals may use different organs to breathe with, they all get oxygen into their bodies through respiration.
Answer:
1. Inhibiting IP3 channels, leading to decreased Ca2 in the sarcoplasm and reduced contraction.
2. Increasing the relative activity of MLCP, leading to a decrease in tension.
3. Activating K channels, increasing K leaking out of the cell which hyperpolarizes it and decreases the likelihood of Ca2 entry.
Explanation
In smooth muscle, cyclic AMP (cAMP) mediates relaxation because cAMP inhibits a specific kinase required for myosin light chain protein (MLCP) phosphorylation, thereby triggering contraction in the smooth muscles. It has been shown that cAMP inhibits 1,4,5-trisphosphate (IP3)-dependent calcium ions (Ca 2+) release by activation of the cGMP-dependent protein kinase (PKG). PKG proteins act to modulate Ca2+ oscillations by stimulating sarcoplasmic Ca2+-ATPase membrane proteins, increasing Ca2+ in the sarcoplasmic reticulum stores and Ca2+ efflux from the cells, and activate voltage-gated potassium (K) channels, thereby leading to membrane hyperpolarization and reducing Ca2+ entry through Ca2+ channels.
The neck is the Cervicis.
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