Answer:
Heart failure
Explanation:
A client with heart failure has decreased cardiac output caused by the heart's decreased pumping ability. A buildup of fluid occurs, causing dyspnea, dependent edema, hepatomegaly, crackles, and jugular vein distention. A client with pulmonary embolism experiences acute shortness of breath, pleuritic chest pain, hemoptysis, and fever. A client with cardiac tamponade experiences muffled heart sounds, hypotension, and elevated central venous pressure. A client with tension pneumothorax has a deviated trachea and absent breath sounds on the affected side as well as dyspnea and jugular vein distention.
Answer:
c. The abnormality is in the thyroid gland itself.
Explanation:
Primary hypothyroidism arises from a pathology in the thyroid gland itself and occurs when the thyroid gland is not able to perform one of its many physiology. Secondary hypothyroidism starts at the level of the pituitary gland and results from under secretion of TSH. TSH is needed to trigger the release of the T3 and T4 stored in the thyroid gland. Tertiary hypothyroidism is caused by a decrease level of the TRH from the hypothalamus. This reduced level, in turn, reduces TSH and thyroid hormone levels in the blood, hence in the body and the patient suffer the symptoms.
For a bloodborne pathogen to be spread, the bodily fluids of an infected person must enter into the bloodstream of another person. The most common cause of transmission in the workplace is when an infected person's blood enters another person's bloodstream through an open wound.
B. exposure to contaminated, abraded skin
The nurse administers cimetidine to a 75-year-old client diagnosed with a Gastric ulcer the nurse should monitor the client for the following adverse reactions Headache, Sleepiness, Confusion, Dizziness, Nausea which contribute for the change in Metal status.
- Cimetidine is a H
Receptor Antagonist. - Gastric partial cells contains receptors for Histamine.
- Histamine is released from the Enterochromaffin cells upon the stimulation from food intake or from Vagus nerve stimulation.
- Thus Histamine released from Enterochromaffin cells binds to H receptors on the parietal cells and increases the secretion of HCl.
- Cimetidine has totally opposite effect and decreases the secretion of HCl from the parietal cells.
- Not only on gastric cells H receptors are also present in CNS where they stimulate brain cells.
- The above point justifies the adverse effects of Cimetidine.
- Patients over the age of 50 or those who are severely ill may experience transient confusion while taking H
blockers, particularly cimetidine.
Hence from these points we can conclude that patients who are above 50 years and receiving Cimetidine medication for gastric ulcer should be monitored for the above mentioned adverse reactions.
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