A is the correct answer.
Hope this helps!
Answer:
two
Explanation:
The endocrine and central nervous systems are the major control systems for regulating homeostasis
Answer:
4. The suspected causative agent must be isolated from the diseased host and grown in pure culture
Explanation:
Robert Koch (1843-1910) was one of the most important bacteriologists of all time. Famous for discovering the tuberculosis bacillus (precisely on March 24, such as today, in 1882), he also discovered the cholera bacillus and is considered the founder of bacteriology. He worked on the isolation of infectious agents and reinfections from pure cultures, experiences from which he established the "Koch Postulates".
These postulates have been taken as a reference that describes the etiology of all the causative agents of an infectious disease, although they were originally used to describe only the tuberculosis bacillus. They are the following:
1- The agent must be present in each case of the disease and absent in the healthy.
2- The agent should not appear in other diseases.
3- The agent must be isolated in a pure culture from the lesions of the disease.
4- The agent has to cause the disease in an animal that can be inoculated
Answer:
Option 3 and 4 are most likely correct
Explanation:
<em>Option 3</em>: We know that area 1 would contain heavy streaking and not single colonies. Therefore, if the loop crosses area 3 and enters area 1, it will definitely result in heavy streaking again.
<em>Option 4</em>: We always need to sterilize properly the loop when streaking in different areas. If we don't do it, it is possible that the loop contains a lot of bacteria that would be streaked again.
Options 1 and 2 are incorrect because, according to question, there is a uniform growth but no isolated colonies. So, contamination or mixed culture would not produce uniform growth streaking rather mixed with other bacterial types.
Answer: DF508 mutation. A Genetic, Hereditary, Autosomal and Recessive Mutation.
Explanation:
Cystic fibrosis (CF) is a recessive autosomal lethal disease, it is most common on Caucasoid populations. Its diagnosis is suggested by the clinical features of chronic obstructive pulmonary disease, persistent pulmonary colonization (particularly with mucoid Pseudomonas strains), meconium ileus, pancreatic insufficiency with or familiarity history of the disease. The FC gene is large, with about 250 Kb of genomic DNA, 27 exons representing about 5% of genomic DNA; encodes a 6.5 kb transcribed mRNA. This mRNA is transcribed into a protein of 1480 amino acid called CFTR (Regulator Transmembrane Conductance Cystic Fibrosis). When a three-base pair deletion, adenosine-thymine-thymine (ATT) identified in the CFTR gene, exon 10, it results in the loss of a single amino acid phenylalanine at position 508 of the protein. This mutation is called DF508; “D” stands for deletion and “F” for phenylalanine amino acid.
