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Sedaia [141]
3 years ago
5

Answer this please thanks

Biology
1 answer:
torisob [31]3 years ago
4 0

Answer:

100%

Answer

Explanation:

I think

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Please

Please yar

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A researcher determines an organism to be eukaryotic, unicellular, and autotrophic. In which taxonomic group should this organis
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According to the characteristics of this organism, it is classified into the Protist kingdom, which brings together unicellular or simple multicellular organisms that do not form tissues, both autotrophic and heterotrophic.

<h3>What is the kingdom protista?</h3>

It includes eukaryote-type organisms that, due to their characteristics, cannot be included in the rest of the kingdoms of this class.

Although most protists are unicellular, there are also multicellular protists and they can have autotrophic or heterotrophic metabolisms.

Therefore, we can conclude that the protist kingdom groups living beings that have cells belonging to the eukaryote group.

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7. A hen's egg with the shell dissolved is placed in a 10% salt solution. Relative
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another man joins each end if the rope Dose this affect the results of the tug of war if not why or why not
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Yes it does because it depends on how strong the man is on either end of the rope
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What is a process whereby an ecosystem can renew itself (points 1?
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Functional connections between chromatin structure, RNA Polymerase II processivity and alternative splicing in Arabidopsis thali
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To find the functional connection's purpose in <em>A. thaliana's</em> chromatin, transcription, and splicing. It was studied and evaluated with the RNAPII processivity (involvement of TFIIS), chromatin structure (roles of BRM, SWI3c, and H1.3 in AS), and spliceosome formation were all investigated at three separate levels (characterization of the role of spliceosome disassembly factor NTR1 in the selection of splice sites).

The alteration of RNAPII elongation rate as the mechanism for NTR1 and TFIIS influences splicing.

Lack of NTR1 causes both localized lower RNAPII levels at those splice locations as well as defective splicing. This can be translated as quicker transcription elongation over those sites in accordance with the kinetic concept of transcription/splicing coupling.

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