Answer:
In one way, the existence of a nonsense mutation would lead to the generation of a premature termination codon that will be identified by the RNA polymerase as a termination sequence encouraged by the activity of Rho factor to dissociate ribosome, thus, discharging RNA polymerase and preventing further transcription mechanism making the transcription of the downstream sequence impossible.
For the second way, there is a need to consider that the mechanism is taking place post-transcriptionally. Thus, the effects should be devised after transcription has taken place and the only fate lies in the mechanism encouraged by the RNA dependent RNA-polymerase. However, for this to take place, the event of genetic recombination can also be taken into account leading to the appearance of the faulty gene in the sequence. Apart from this, the open reading frame is required to be co-expressive that would be the most suitable factor, which determines whether the downstream sequences will be transcribed or not post nonsense mutation.
However, the total change relies upon the fact that the mutation is taking place artificially or is induced naturally. One more thing to consider is that there is an existence of another gene known as MCB 354, which is encrypted by another gene and is probably monitored by another promoter sequence. Thus, co-expression would probably be the mechanism in terms of the rho-dependent termination.
During anaphase, each pair of chromosomes is separated into two identical, independent chromosomes. The chromosomes are separated by a structure called the mitotic spindle. ... The separated chromosomes are then pulled by the spindle to opposite poles of the cell.
Hope that helps
A recessive trait for the yellow gene found in the population of red squirrelfish.
The species which reproduce s_xually will more likely survive. Because they produce genetic variation in the offspring, the desease will less likely to affect all the individuals in a population.
Might be Cell Differentiation...