Red tide is an explosive growth of the <span>dinoflagellates bacteria. </span>
Answer:
The equivalent magnetization (EM) and mantle Bouguer anomaly (MBA) were calculated along the ultraslow-spreading Mohns Ridge axis in the Norwegian-Greenland Sea. The magnetic anomaly and the associated EM were compared with the bathymetry, MBA, seismically determined crustal structure and geochemical data at both the inter-segment scale (>60 km) and the intra-segment scale (20–60 km). At the inter-segment scale, the magnetic highs at the segment centers are independent of the MBA. Of the 13 segments, 9 with magnetic anomalies >700 nT coincide with axial volcanic ridges identified from multibeam bathymetry maps, which suggests that the magnetic highs at the segment centers may be more associated with the extrusive lavas rather than the amount of magma supply. With few exceptions, the magnetic anomaly lows associated with MBA highs at the segment ends increase from south to north. This trend might be explained by thickened extrusive basalts and/or more serpentinized peridotites at the segment ends in the north. At the intra-segment scale, the most prominent features are the decreases in the magnetic anomalies and associated EMs from the segment centers to the ends. The intra-segment magnetic anomalies have positive and negative correlations with the bathymetry and MBA, respectively. The magnetic signal modeled by the seismically determined layer 2A with an assumed constant magnetization is remarkably consistent with the observed magnetic anomaly, which strongly suggests that the thickness of the extrusive basalts dominates the magnetic structure in each segment along the Mohns Ridge. In general, the thickness of the extrusive basalts dominates the magnetic structure along the Mohns Ridge, whereas the contributions from serpentinized peridotites may be significant at the segment ends and may produce long-wavelength magnetic variations. The magnetic data can be used as an indicator of the thickness of the extrusive basalts within segments along the ultraslow-spreading Mohns Ridge.
Explanation:
Answer:
Sickle cell disease in an autosomal recessive inherited disease which is caused by the mutation in the HBB (hemoglobin-β gene) gene present on the chromosome no. 11. In sickle cell, the red blood cells become sickle shape due to the abnormal shape of hemoglobin present in it.
These sickle cells are known to form a blockage in the blood vessels thereby causing damage to the vital organs. Human spleen constantly destroys sickle cells because they get trapped in it which causes a disease called sickle cell anemia.
Sickle cell anemia is normally the problem in genetic disease which is inherited from the parents to children. When both the DNA strand has a mutation in their HBB gene then only this disease will affect the individual.
Answer:
Histidine- Proline- Tryptophan- Stop codon
Explanation:
The mRNA strand in the question is produced as a result of a process called TRANSCRIPTION. However, the mRNA transcript is further used to synthesize an amino acid sequence in a process called TRANSLATION. The mRNA sequence is read in a group of three nucleotide bases called CODON.
Each codon specifies an amino acid which is represented in the GENETIC CODE. In this case, a sequence of mRNA strand is given as: CACCCAUGGUGA
The following codons in the sequence specify the following amino acid:
CAC - Histidine
CCA - Proline
UGG - Tryptophan
UGA - Stop codon
Hence, the amino acid sequence encoded by the given mRNA strand is Histidine- Proline- Tryptophan- Stop codon.
N.B: After a stop codon (UGA, UAA, UAG) is encountered, the translation process stops.
