It could cause liver damage and problems to the body from the medication of depression
Answer:
The Living Will
Explanation: Living Will is a document giving directives to the care givers and physicians on how and when to withdraw medical supports to patients with terminally illness or withhold artificial supports.
It can only be used if the person will not be able to give the instructions himself,without this document the doctor is legally and ethical bound to keep the terminally patient alive till the Document is fully signed.The living will help the terminally ill person and his or her relatives to know about what is to be administered on the person.
Answer:
The correct option is : a. serous fluid in the pericardial cavity
Explanation:
The heart is enclosed in a sac containing the heart, known as the pericardium. The pericardium has two layers- fibrous and serous layer.
The serous layer of the pericardium produces and secretes the pericardial fluid into the pericardial cavity.
The pericardial fluid is a lubricating serous fluid present in the pericardial cavity. It reduces the friction caused between the pericardial membranes by lubricating the epicardial surface.
Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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