Answer:
Hey there the answer would be D. Keratin is the pigment responsable for skin color.
Explanation:
A. The top layer of the epidermus is made of dead skin cells. The inner player has to be constatnly making more cells
B.The body procudes more melanin with stronger sunlight exposure. This results because the UV of the sun causes T-T dimers which is a type of DNA damage.
C. UV causes genetic mutation which can result in cancer if not fixed by the cell
D.Keratin is found in the epidermis and with thick layer of skin found in the hands and feet. Melainin is responsible for skin color.
Answer:
Living things may be distinguished from non-living things in their ability to carry on life processes such as movement, respiration, growth, responsiveness to environmental stimuli and reproduction. This view of living may be appropriate at this age but has some limitations and can lead to the alternative conceptions above. For example movement in plants is not apparent to students and consequently they may not consider plants living.
If Gabriel developed diarrhea as a result of taking an antacid, he's been taking an antacid that contains magnesium.
Magnesium will act as a laxative.
Answer:
indeterminate
Explanation:
The answer would be <u>an indeterminate number of generations.</u>
The blue trait is a heterozygous trait. In other words, the blue allele does not really exist. Two different alleles team up to produce the trait, whereas, only two the same allelic conditions can produce a true-breeding flock.
<em>Hence, it will take an indeterminate number of generations to produce a true-breeding flock of the Blue Andalusions.</em>
Answer:
on a tyrosine residue
bind to insulin
Explanation:
The insulin receptor substrate-1 (IRS1) is a signaling protein that can be phosphorylated on multiple tyrosine and serine/threonine residues. IRS1 contains several conserved domains including a pleckstrin (PH) domain and a PTB domain involved in protein phosphorylation and ligand binding. In the first place, IRS1 is phosphorylated on a tyrosine residue, and then IRS1 binds to insulin or the insulin-like growth factor-1 (IGF-1), thereby activating transduction pathways such as, for example, MAPK/ERK. Moreover, RS1 is also phosphorylated on serine residues, thereby triggering opposite effects in insulin-associated signaling.