The above-mentioned method is used to analyze the streak of a mineral. Streak can be different from that of the color of the mineral, itself. It is used for soft minerals, and such minerals have their characteristic streaks. So the correct answer to your question is D.
Answer:
No, glucose and sucrose are not complex substances. Both are simple substances. Complex molecules are polysaccharides such as glycogen and starch.
Explanation:
Glucose is made of one sugar atom and known as monosaccharide, while sucrose is composed of two sugar atoms i. e. glucose and fructose. So that's why glucose and sucrose are not complex molecules.
Answer:
A) 300
B) 3:1
C) 9 long yellow : 3 long green : 3 short yellow : 1 short green
Explanation:
Long stems (L_) are dominant to short stems (ll)
Yellow seeds (Y_) are dominant to green seeds (yy)
We interbred pea plants with long stems and yellow seeds (L_Y_), but they had a short green parent (llyy) that could have only produced <em>ly</em> gametes, so our plants are heterozygous <em>LlYy</em>.
C) We interbred them LlYy x LlYy. If the two genes are unlinked, this is a typical dihybrid cross and from Mendel's law of independent assortment we know that the offspring will have the following phenotypic ratios:
- 9/16 L_Y_ (Long, yellow)
- 3/16 L_yy (Long, green)
- 3/16 llY_ (short, yellow)
- 1/16 llyy (short, green)
A) 3/16 × 1600 = 300 plants will be long and green.
B)
9/16 + 3/16 = 12/16= 3/4 plants will be yellow;
3/16 + 1/16 = 4/16= 1/4 plants will be green.
The ratio will be 3 yellow : 1 green
Answer:
The correct insulin pathway is described as under:
2. Binding of insulin to the alpha subunit of the insulin receptor
8. Activation of insulin receptor tyrosine kinase
3. Phosphorylation of IRS proteins
6. Phosphorylation of phosphinositide 3-kinase (PI-3K)
4. Conversion of PIP2 to PIP3
7. Activation of PIP3-dependent protein kinase B (PDK1)
5. Glut4 receptors transported to the cell membrane
Explanation:
The insulin signaling pathway is described as under:
RTK (receptor tyrosine kinases) which is a receptor for insulin is an extracellular receptor but in contrast to other cell surface receptors it is catalytic in nature. In the absence of insulin (ligand), it is monomeric but as soon as it gets activated (activation occurs upon ligand binding), it undergo dimerization. It leads to auto-phosphorylation in it's tyrosine residue which subsequently leads to phosphorylation of tyrosine residue of other receptors. Such hyper-phosphorylated receptor have high affinity with enzyme/molecule like IRS protein which have SH2 domain . IRS down stream activates phosphinositide 3-kinase (PI-3K). This enzyme converts component of animal cell membrane PIP2 into PIP3. PIP3 also remains membrane bound but it has the potential to phosphorylate another enzyme named as PIP3-dependent protein kinase B (PDK1). Further, PDK1 leads to the activation of Akt or PK-B. Akt is a serine-threonine kinase which ultimately leads to the recruitment of Glut4 receptors on cell membrane for uptake of more and more glucose into the cell.
Note: Apart from this Akt also phosphorylates another protein named as FOXO which ultimately causes cell growth, Akt can also phosphorylate BAD protein so as to restrict cell apoptosis or we can say it leads to cell survival, Akt also leads to translation in a cell with the help of mTOR raptor etc.
