I believe the answer is B predicts impact on coastal populations
please let me know if I'm incorrect
D. The macronucleus does not disintegrate during conjugation.
The macronucleus contains the working copies of genes needed to support an enormous cell. It is formed by a division of the fused diploid nucleus after conjugation followed by multiple rounds of internal gene replication which does not occurs in the micronucleus.
Answer:
He can deduce that The pO2 of her cells is less than or equal to 200 mmHg.
Explanation:
Partial pressure of oxygen(pO2) measures the amount of gaseous oxygen dissolved in the blood. It purposes to measure the effectiveness of the lungs in pulling oxygen from the atmosphere to the lungs.
In the incubator where the gases levels are carefully regulated, a pO2 of 200mmHg on the incubator deduces that the pO2 of her cells is equal or less than 200mmHg.
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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